1. Convexity meningiomas are some of the most surgically accessible meningiomas, so we can usually remove them (resection) completely

2. Centrioles were observed in angiomatous meningiomas, but cilia and other ciliary structures were found only in the endotheliomatous and fibroblastic meningiomas.

3. Circumferential intradural en plaque meningiomas are, however, rare

4. There was no mortality among patients with atypical meningioma, while four out of five patients with anaplastic meningioma died during follow-up.

5. The histological classification used was that of Courville (1950) and Rubinstein (1972), but “angioblastic” meningiomas were segregated into 3 groups: highly vascularized meningiomas, hemangioblastomas, and hemangiopericytomas.

6. The tumour showed features of a meningioma and a rhabdoid morphology with angiomatous components and was considered to be a rhabdoid meningioma. After surgery a small residual tumour remained.

7. Morphologically, “angioblastic meningioma” and “hemangiopericytoma of the meninges” show striking common features.

8. Differential diagnosis has to consider angioblastic meningioma, haemangiopericytoma, and hypernephroma metastasis.

9. All three tumors were primary diagnosed as meningiomas (one: vascular, two angioblastic).

10. In two meningiomas the alkaline phosphatase activity was confined to mutated cell clones.

11. Convexity meningiomas are tumors that grow on the surface of the brain (called the Convexity)

12. Finally, the possibility was raised that among (malignant) angioblastic meningiomas also haemangiopericytomas are present.

13. Objective To summarize the experience of pterional craniotomy microsurgical resection of giant tuberculum sellae meningiomas.

14. By using 3D-MRI in almost all patients undergoing radiotherapy of a meningioma, tumor shrinkage is detected.

15. Diagnosed with meningioma, a type of brain tumor, Cheng was in desperate need of surgery.

16. It is especially important to differentiate these lesions from the angioblastic meningiomas because they are common supratentorially.

17. Endotheliomatous and transitional forms constituted 85% of the total (71.5% of intracranial tumors), fibroblastic forms 6.6 and 7.5%, respectively, and highly vascularized (endotheliomatous or transitional) meningiomas 5.2% of the intracranial tumors, while true “angioblastic” meningiomas (hemangioblastomas and hemangiopericytomas) amounted to 2.8% of the total (3.1% of the intracranial tumors). 1.2% were “atypical” (so-called malignant) meningiomas; true meningeal sarcomas were excluded.

18. The occurrence of cilia and related structures were studied electron-microscopically in 14 endotheliomatous, 3 fibroblastic, and 3 angiomatous meningiomas.

19. We compared the number of nucleoli in cells of meningioma cultures which have lost none, one, two or more acrocentric chromosomes.

20. 11 Conclusion Homogeneous enhancement, visibility of the pituitary gland, the dural tail sign and compression of the carotid artery are the characteristics of the intrasellar meningioma.

21. The other tumour, an angioblastic meningioma, had a hyperdiploid stemline with 47 chromosomes including a minute marker, and a closely related 48-chromosome sideline with 2 minute markers of different size.

22. Furthermore, clinical data and growth pattern of “angioblastic meningioma” and “hemangiopericytoma of the meninges” are well comparable. Therefore, it seems to be justified to interpret these tumors as a tumor entity with identical histogenesis.

23. Aims: Despite the important prognostic value of brain invasion in meningiomas, little attention has been paid to its massessment, and the parameters associated with brain invasion Assessability (identification of brain tissue in the surgical specimen) are not well characterized

24. To control hemodynamic disturbances during removal of cerebral meningiomas a successful application finds a method of preliminary embolism of afferent vessels of the newgrowth Alimenting it from the external carotid system with polystyrene emboli and of direct blood transfusion.

25. They are optimally treated with gross-total resection; partial resections may result in tumor recurrence within 5 postoperative years.[ 1 , 3 , 7 ] Here, we present a 40-year-old female with T6-T7 Circumferential meningioma whose residual anterior disease became symptomatic 5

26. The clinical presentation together with macroscopic and microscopic findings in a case of epidural fronto-parasagittal hemangiopericytoma occurring in a 59 years old female are reported. Pathogenesis, clinical and macroscopic findings, and microscopicaspects of this tumor aspected with the angioblastic (angiomatous) meningioma are discussed.

27. THE SECOND TYPE (NF-2) is characterized by the occurrence of acoustic neuromas, usually bilateral, as well as meningiomas and other tumours of the nervous system. Café-au-lait spots and cutaneous neurofibromas may be seen, but are usually few in number and much less common than in NF-1.

28. The chromosomal investigation of a series of 40 short term cultured human meningiomas revealed as main finding the loss of a short acrocentric chromosome of the 21–22 group in 32 tumors. 6 tumors had an apparantly normal chromosome complement, 2 tumors had a numerical chromosome aberration without G-chromosome loss involved.

29. Pituitary Adenomas arise from the pituitary gland and are by far the most common growth associated with the gland; they account for 15% of primary brain tumors and are the third most common intracranial tumor after meningiomas and gliomas.The great majority (over 90%) of pituitary Adenomas are benign slow growing tumors, approximately 5-10% are somewhat …

30. By reviewing the operative history of 20 meningioma cases displaying midline subfrontal and presellar localization, the author found that in 5 cases the growth was attached to the lamina cribrosa and the crista galli, in 3 cases it was attached adjacent to the tuberculum sellae, but spreading onto the limbus sphenoidale also; in 12 cases it was found adhering to the sphenoid plane.

31. Based on the profiles of aldolase isozyme, the tumours tested seemed to be classified into two groups: those with a higher activity of aldolase C in A-C set (astrocytomas such as mixed type with oligodendroglioma, plump cell type and somewhat anaplastic type) and those with a rather low to no detectable activity of aldolase C in A-C set (malignant glioma, neuroblastoma, neurinoma, meningioma, pituitary adenoma, pinealoma, craniopharyngioma, secondary carcinoma).